Critical Points in Tumorigenesis: A Carcinogen-Initiated Phase Transition Analyzed via Single-Cell Proteomics.
Supramolecular Probes for Assessing Glutamine Uptake Enable Semi-Quantitative Metabolic Models in Single Cells.
JAK-STAT Pathway Activation in Malignant and Non-Malignant Cells Contributes to MPN Pathogenesis and Therapeutic Response.
Analysis of Single-Cell Secretion Reveals a Role for Paracrine Signaling in Coordinating Macrophage Response to TLR4 Stimulation.
Highly Multiplexed Profiling of Immune Effector Functions Reveals Deep Functional Heterogeneity in Response to Pathogenic Ligands.
Glioblastoma Cellular Architectures are Predicted Through the Characterization Of Two-Cell Interactions.
Human NK Cells Licensed by Killer Ig Receptor Genes have an Altered Cytokine Program that Modifies CD4+ T Cell Function.
Conversion of Danger Signals into Cytokine Signals by Hematopoietic Stem and Progenitor Cells for Regulation of Stress-Induced Hematopoiesis.